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Chemical Chromatin Interactions Research Core

The goals of the Research Core are to enhance human health by translating new knowledge of chemical-chromatin interactions into the public health and clinical research arenas.
The composition of the Chemical-Chromatin Interactions Research Core reflects biology-driven science coupled with various aspects of the biological consequences of the interaction of chemicals with chromatin.
   
 
Faculty within this Research Core are actively extending their basic chemicals and chromatin research programs into the public health and clinical research concerning cancer development and control.
   
 
2004 publications by members of the core. For a complete list of publications click here.
Chen, Q. M., Alexander, D., Morrissy, S., Terrang, J., Sun, J. P., Xie, L. F., and Purdom, S. (2004) Corticosteroids Prevent Doxorubicin Induced Apoptosis in Cardiomyocytes, Submitted.
Yoon, H.S, Ramachandiran, S., Chacko, M.A.S., Monks, T. J., and Lau, S. S. Tuberous sclerosis-2 tumor suppressor modulates ERK and B-Raf activity in transformed renal epithelial cells. Am. J. Physiol. Renal Physiol. 286: F417-424, 2004.
Watts, G. S., Oshiro, M. M., Junk, D. J., Wozniak, R. J., Watterson, S., Domann, F. E., and Futscher, B. W. (2004). The acetyltransferase p300/CBP-associated factor (PCAF) is a p53 target gene in breast tumor cells. Neoplasia, In Press
Wood, J., Terrand, J., Sun, H. P., and Chen, Q. M. (2004). c-Fos Protein Phosphorylation Contributes to Increased Stability of c-fos and AP-1 Activation in Oxidant Response of Cardiomyocytes., Submitted.


Links to our current news from the Research Core:

Southwest Environmental Health Sciences Center
University of Arizona College of Pharmacy, Room 244
PO Box 210207, Tucson, AZ, USA  85721-0207
swehsc-info@pharmacy.arizona.edu
520-626-5594
520-626-6944(FAX)

Funded by NIEHS grant # ES06694

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Last update: March 1, 2005 		Page Content: Travis Biazo
Web Master: Mike Kopplin